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chip grade antibodies against p creb  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc chip grade antibodies against p creb
    Figure 8. <t>CREB</t> bound with AnkG promoter to regulate AnkG transcription. A-B) The expression level of p-Akt/T-Akt in hippocampus from SSTR3−/−
    Chip Grade Antibodies Against P Creb, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 97/100, based on 2047 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/chip+grade+antibodies+against+p+creb/pm39804991-856-12-17?v=Cell+Signaling+Technology+Inc
    Average 97 stars, based on 2047 article reviews
    chip grade antibodies against p creb - by Bioz Stars, 2026-07
    97/100 stars

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    1) Product Images from "The Primary Cilia are Associated with the Axon Initial Segment in Neurons."

    Article Title: The Primary Cilia are Associated with the Axon Initial Segment in Neurons.

    Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

    doi: 10.1002/advs.202407405

    Figure 8. CREB bound with AnkG promoter to regulate AnkG transcription. A-B) The expression level of p-Akt/T-Akt in hippocampus from SSTR3−/−
    Figure Legend Snippet: Figure 8. CREB bound with AnkG promoter to regulate AnkG transcription. A-B) The expression level of p-Akt/T-Akt in hippocampus from SSTR3−/−

    Techniques Used: Expressing

    Figure 9. CREB expression restored AnkG structure and plasticity in SSTR3−/−neurons. A-B) Neurons in forskolin condition possessed longer AIS and AIS was shorter with 666-15 and CREB siRNA treatments. A. Immunocytochemistry results for AnkG in the SSTR3+/+ neurons in control, forskolin, 666- 15 and CREB siRNA treatments. MAP2 and GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. B. Statistical graph of AIS length with different treatments. Left, One-way ANOVA followed by Dunnet post-hoc test was based on data from three independent experiments; F (2, 146) = 55.51; control versus forsklin, p < 0.001; control versus 666-15, p < 0.001. control, n = 53; forskolin, n = 38; 666-15, n = 58. Right, Statistical graph of AIS length with CREB siRNA expression. p < 0.001; control, n = 35; CREB siRNA, n = 41. Unpaired t test, data from 3 independent experiments. The results were represented by mean ± SEM. C-D) AIS length recovered in SSTR3−/−neurons with forskolin treatments. C. Immunocytochemistry results for AnkG in the SSTR3−/−neurons in control and forskolin condition. MAP2 (green) indicated dendrites and soma, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. D. Statistical graph of AIS length with forskolin treatments. One-way ANOVA followed by Tukey post-hoc test was based on data from three independent experiments; F (2, 97) = 21.69; Control versus forskolin 24 h, p = 0.002; Control versus forskolin 48 h, p < 0.001; forskolin 24 h versus forskolin 48 h, p = 0.005. control, n = 43; forskolin 24 h, n = 33; forskolin 48 h, n = 24. The results were represented by mean ± SEM. E-F) CREB cDNA expression restored AIS length in SSTR3−/−neurons. E. Immunocytochemistry results for AnkG in the SSTR3−/−neurons with control and CREB cDNA. GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and
    Figure Legend Snippet: Figure 9. CREB expression restored AnkG structure and plasticity in SSTR3−/−neurons. A-B) Neurons in forskolin condition possessed longer AIS and AIS was shorter with 666-15 and CREB siRNA treatments. A. Immunocytochemistry results for AnkG in the SSTR3+/+ neurons in control, forskolin, 666- 15 and CREB siRNA treatments. MAP2 and GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. B. Statistical graph of AIS length with different treatments. Left, One-way ANOVA followed by Dunnet post-hoc test was based on data from three independent experiments; F (2, 146) = 55.51; control versus forsklin, p < 0.001; control versus 666-15, p < 0.001. control, n = 53; forskolin, n = 38; 666-15, n = 58. Right, Statistical graph of AIS length with CREB siRNA expression. p < 0.001; control, n = 35; CREB siRNA, n = 41. Unpaired t test, data from 3 independent experiments. The results were represented by mean ± SEM. C-D) AIS length recovered in SSTR3−/−neurons with forskolin treatments. C. Immunocytochemistry results for AnkG in the SSTR3−/−neurons in control and forskolin condition. MAP2 (green) indicated dendrites and soma, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. D. Statistical graph of AIS length with forskolin treatments. One-way ANOVA followed by Tukey post-hoc test was based on data from three independent experiments; F (2, 97) = 21.69; Control versus forskolin 24 h, p = 0.002; Control versus forskolin 48 h, p < 0.001; forskolin 24 h versus forskolin 48 h, p = 0.005. control, n = 43; forskolin 24 h, n = 33; forskolin 48 h, n = 24. The results were represented by mean ± SEM. E-F) CREB cDNA expression restored AIS length in SSTR3−/−neurons. E. Immunocytochemistry results for AnkG in the SSTR3−/−neurons with control and CREB cDNA. GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and

    Techniques Used: Expressing, Immunocytochemistry, Control



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    Figure 8. <t>CREB</t> bound with AnkG promoter to regulate AnkG transcription. A-B) The expression level of p-Akt/T-Akt in hippocampus from SSTR3−/−
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    Image Search Results


    Figure 8. CREB bound with AnkG promoter to regulate AnkG transcription. A-B) The expression level of p-Akt/T-Akt in hippocampus from SSTR3−/−

    Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

    Article Title: The Primary Cilia are Associated with the Axon Initial Segment in Neurons.

    doi: 10.1002/advs.202407405

    Figure Lengend Snippet: Figure 8. CREB bound with AnkG promoter to regulate AnkG transcription. A-B) The expression level of p-Akt/T-Akt in hippocampus from SSTR3−/−

    Article Snippet: Precleared chromatin supernatants were subjected to immunoprecipitation at 4 °C overnight using ChIP-grade antibodies against p-CREB (9198, Cell Signaling Technology).

    Techniques: Expressing

    Figure 9. CREB expression restored AnkG structure and plasticity in SSTR3−/−neurons. A-B) Neurons in forskolin condition possessed longer AIS and AIS was shorter with 666-15 and CREB siRNA treatments. A. Immunocytochemistry results for AnkG in the SSTR3+/+ neurons in control, forskolin, 666- 15 and CREB siRNA treatments. MAP2 and GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. B. Statistical graph of AIS length with different treatments. Left, One-way ANOVA followed by Dunnet post-hoc test was based on data from three independent experiments; F (2, 146) = 55.51; control versus forsklin, p < 0.001; control versus 666-15, p < 0.001. control, n = 53; forskolin, n = 38; 666-15, n = 58. Right, Statistical graph of AIS length with CREB siRNA expression. p < 0.001; control, n = 35; CREB siRNA, n = 41. Unpaired t test, data from 3 independent experiments. The results were represented by mean ± SEM. C-D) AIS length recovered in SSTR3−/−neurons with forskolin treatments. C. Immunocytochemistry results for AnkG in the SSTR3−/−neurons in control and forskolin condition. MAP2 (green) indicated dendrites and soma, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. D. Statistical graph of AIS length with forskolin treatments. One-way ANOVA followed by Tukey post-hoc test was based on data from three independent experiments; F (2, 97) = 21.69; Control versus forskolin 24 h, p = 0.002; Control versus forskolin 48 h, p < 0.001; forskolin 24 h versus forskolin 48 h, p = 0.005. control, n = 43; forskolin 24 h, n = 33; forskolin 48 h, n = 24. The results were represented by mean ± SEM. E-F) CREB cDNA expression restored AIS length in SSTR3−/−neurons. E. Immunocytochemistry results for AnkG in the SSTR3−/−neurons with control and CREB cDNA. GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and

    Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

    Article Title: The Primary Cilia are Associated with the Axon Initial Segment in Neurons.

    doi: 10.1002/advs.202407405

    Figure Lengend Snippet: Figure 9. CREB expression restored AnkG structure and plasticity in SSTR3−/−neurons. A-B) Neurons in forskolin condition possessed longer AIS and AIS was shorter with 666-15 and CREB siRNA treatments. A. Immunocytochemistry results for AnkG in the SSTR3+/+ neurons in control, forskolin, 666- 15 and CREB siRNA treatments. MAP2 and GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. B. Statistical graph of AIS length with different treatments. Left, One-way ANOVA followed by Dunnet post-hoc test was based on data from three independent experiments; F (2, 146) = 55.51; control versus forsklin, p < 0.001; control versus 666-15, p < 0.001. control, n = 53; forskolin, n = 38; 666-15, n = 58. Right, Statistical graph of AIS length with CREB siRNA expression. p < 0.001; control, n = 35; CREB siRNA, n = 41. Unpaired t test, data from 3 independent experiments. The results were represented by mean ± SEM. C-D) AIS length recovered in SSTR3−/−neurons with forskolin treatments. C. Immunocytochemistry results for AnkG in the SSTR3−/−neurons in control and forskolin condition. MAP2 (green) indicated dendrites and soma, AnkG (red) marked AIS, and DAPI (blue) marked the nuclei. Scale bar, 10 μm. D. Statistical graph of AIS length with forskolin treatments. One-way ANOVA followed by Tukey post-hoc test was based on data from three independent experiments; F (2, 97) = 21.69; Control versus forskolin 24 h, p = 0.002; Control versus forskolin 48 h, p < 0.001; forskolin 24 h versus forskolin 48 h, p = 0.005. control, n = 43; forskolin 24 h, n = 33; forskolin 48 h, n = 24. The results were represented by mean ± SEM. E-F) CREB cDNA expression restored AIS length in SSTR3−/−neurons. E. Immunocytochemistry results for AnkG in the SSTR3−/−neurons with control and CREB cDNA. GFP (green) indicated morphology of neurons, AnkG (red) marked AIS, and

    Article Snippet: Precleared chromatin supernatants were subjected to immunoprecipitation at 4 °C overnight using ChIP-grade antibodies against p-CREB (9198, Cell Signaling Technology).

    Techniques: Expressing, Immunocytochemistry, Control